What do we REALLY see in the article below?---We see an extremely complex DESIGNED system (paragraph 2) that appears to be flawless....until twisting of the strands cause problems (paragraph 3). We see a finely tuned, DESIGNED system under the stress of sinful generations breaking down. (Like all designed systems, from a car to a cell phone).
1 "Scientists have revealed for the first time the molecular steps that turn on bacteria's pathogenic genes. Using an array of high-powered X-ray imaging techniques, the researchers at the Department
of Energy's Lawrence Berkeley National Laboratory (Berkeley Lab) showed that histone-like proteins that bind to DNA are related to the physical twisting of the genetic strand, and that the supercoiling of the chromosome can trigger the expression of genes that make a microbe invasive.
2 The researchers looked at how the long strands of DNA are wound tight, a necessity if they are to fit into compact spaces. For eukaryotes, the strands wrap around histone proteins to fit inside a nucleus. For single-celled prokaryotes, which include bacteria, HU proteins serve as the histones, and the chromosomes bunch up in the nucleoid, which lacks a membrane.
3 When the normal twists and turns of DNA compaction turn into supercoiling, trouble can begin.
4 "We needed the interaction of these different techniques to get the overall picture of how the HU interactions with DNA were affecting the bacteria," added Larabell, who is also a professor of anatomy at UC San Francisco. "With X-ray tomography, we're able to see the natural contrast in organic material in as close to a living state as possible, and we can provide quantitative comparisons of how compacted the chromosomes were in pathogenic and normal strains of E. coli." Phys.Org
For we know that the whole creation groaneth
and travaileth in pain together until now.
Romans 8:22
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| What was in that TREE of Life to keep this from happening? hmmmm |
2 The researchers looked at how the long strands of DNA are wound tight, a necessity if they are to fit into compact spaces. For eukaryotes, the strands wrap around histone proteins to fit inside a nucleus. For single-celled prokaryotes, which include bacteria, HU proteins serve as the histones, and the chromosomes bunch up in the nucleoid, which lacks a membrane.
3 When the normal twists and turns of DNA compaction turn into supercoiling, trouble can begin.
4 "We needed the interaction of these different techniques to get the overall picture of how the HU interactions with DNA were affecting the bacteria," added Larabell, who is also a professor of anatomy at UC San Francisco. "With X-ray tomography, we're able to see the natural contrast in organic material in as close to a living state as possible, and we can provide quantitative comparisons of how compacted the chromosomes were in pathogenic and normal strains of E. coli." Phys.Org
