"The “Hobbits” (Homo floresiensis), also known as “Flores Man,” are an extinct human people group once theorized by some researchers to be an evolutionary transition toward modern humans. This was partly because, like Australopithecus sediba, they appeared to show a unique combination of both primitive and advanced human-like features.
However, many paleoanthropologists now regard Homo floresiensis as anatomically human individuals affected by genetic mutations or environmental factors associated with life on small islands—a phenomenon known as insular dwarfism. These mutations are thought to have slowed the growth of both their bodies and brains.
Partial Hobbit skeletons, including leg bones, hands, feet, a partialpelvis, and one complete skull from 15 individuals, were recovered in 2003. The Hobbits inhabited the small Indonesian island of Flores. They stood about 3 feet 6 inches tall, and their skull volume was roughly one-third that of an average modern human. They were considerably shorter than even the smallest modern human populations, such as the Mbenga and Mbuti tribes, whose average adult height is about 4 feet 11 inches.
Nonetheless, despite the modern human features, some still argue for an evolutionary link, arguing that the evidence “strongly supports the idea that H. floresiensis evolved from H. erectus on Java.” In a press release in Science by Bridget Alex last year, evolutionists admit that
"....the exact lineage [of the Hobbits] is debated. The most prominent theory is that they evolved from a larger hominin, like Homo erectus, that became isolated on the Indonesian island of Flores and underwent a process called island dwarfism, becoming progressively smaller due to limited resources. However, some recent studies have challenged this theory, suggesting they may have evolved from a more primitive, pre-erectus ancestor."
This speculation argues that the Hobbits represent a “missing link” in human evolution that eventually stopped evolving, surviving for ages in an unchanged form. In the words of Peter Brown, a paleoanthropologist at the University of New England in Australia, and archaeologist Michael Morwood, they were a “previously undiscovered species of archaic human that had survived for thousands of years after the Neanderthals had died out.”
The latest research by paleoanthropologists Monson and Weitz, presented in The Conversation on 24 September, argues that the small stature of the Hobbits from Flores was due to a slowdown in growth that occurred during childhood. In other words, their size was likely an anomaly caused by abnormal hormonal variations that can be explained biologically.
Specifically, their reduced size is thought to have resulted from disruptions in the regulation of human growth hormones. These growth hormones (GH) include GH-binding protein (GHBP) and insulin growth factor-I (IGF-I).
Still wanting to Darwinize these humans, evolutionists are trying a reassessment of certain evolutionary assumptions:
"Until Homo floresiensis was discovered, scientists assumed that the evolution of the human lineage was defined by bigger and bigger brains. Via a process called encephalization, human brains evolved to be relatively more massive than would be expected based on corresponding body size. This proportionally bigger brain is what anthropologists argued enabled us and our relatives to perform more complex tasks such as using fire, forging and wielding tools, making art, and domesticating animals. But these theories had to be thrown out the window when archaeologists announced our fossil cousins Homo floresiensis via scientific publication in 2004. Homo floresiensis lived from about 700,000 to 60,000 years ago in the rainforests of Indonesia, partially contemporaneous with our own species."
Despite the many evolutionary claims made about Homo floresiensis, the Pygmies, Homo luzonensis (from the Philippines), and other examples, the traits of well-proportioned small humans can be fully explained biologically. The evolutionary speculations largely serve as a distraction.
The Hobbits’ reduced stature appears to result from slowed growth during childhood caused by variations in hormone regulation —specifically the growth hormones (GH) which include GH-binding protein (GHBP) and insulin growth factor-I (IGF-I)."
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